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.1993 Mar 1;90(5):1977-81.
doi: 10.1073/pnas.90.5.1977.

Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease

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Apolipoprotein E: high-avidity binding to beta-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease

W J Strittmatter et al. Proc Natl Acad Sci U S A..

Abstract

Apolipoprotein E is immunochemically localized to the senile plaques, vascular amyloid, and neurofibrillary tangles of Alzheimer disease. In vitro, apolipoprotein E in cerebrospinal fluid binds to synthetic beta A4 peptide (the primary constituent of the senile plaque) with high avidity. Amino acids 12-28 of the beta A4 peptide are required. The gene for apolipoprotein E is located on chromosome 19q13.2, within the region previously associated with linkage of late-onset familial Alzheimer disease. Analysis of apolipoprotein E alleles in Alzheimer disease and controls demonstrated that there was a highly significant association of apolipoprotein E type 4 allele (APOE-epsilon 4) and late-onset familial Alzheimer disease. The allele frequency of the APOE-epsilon 4 in 30 random affected patients, each from a different Alzheimer disease family, was 0.50 +/- 0.06; the allele frequency of APOE-epsilon 4 in 91 age-matched unrelated controls was 0.16 +/- 0.03 (Z = 2.44, P = 0.014). A functional role of the apolipoprotein E-E4 isoform in the pathogenesis of late-onset familial Alzheimer disease is suggested.

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References

    1. Arteriosclerosis. 1983 Jul-Aug;3(4):310-5 - PubMed
    1. Proc Natl Acad Sci U S A. 1985 Jan;82(1):203-7 - PubMed
    1. Proc Natl Acad Sci U S A. 1986 Feb;83(4):1125-9 - PubMed
    1. Proc Natl Acad Sci U S A. 1986 Feb;83(4):1130-4 - PubMed
    1. Biochim Biophys Acta. 1987 Jan 13;917(1):148-61 - PubMed

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