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Review
.2022 Mar 15;13(3):185-202.
doi: 10.4239/wjd.v13.i3.185.

Maternal low protein diet and fetal programming of lean type 2 diabetes

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Review

Maternal low protein diet and fetal programming of lean type 2 diabetes

Vidyadharan Alukkal Vipin et al. World J Diabetes..

Abstract

Maternal nutrition is found to be the key factor that determines fetal healthin utero and metabolic health during adulthood. Metabolic diseases have been primarily attributed to impaired maternal nutrition during pregnancy, and impaired nutrition has been an immense issue across the globe. In recent years, type 2 diabetes (T2D) has reached epidemic proportion and is a severe public health problem in many countries. Although plenty of research has already been conducted to tackle T2D which is associated with obesity, little is known regarding the etiology and pathophysiology of lean T2D, a variant of T2D. Recent studies have focused on the effects of epigenetic variation on the contribution ofin utero origins of lean T2D, although other mechanisms might also contribute to the pathology. Observational studies in humans and experiments in animals strongly suggest an association between maternal low protein diet and lean T2D phenotype. In addition, clear sex-specific disease prevalence was observed in different studies. Consequently, more research is essential for the understanding of the etiology and pathophysiology of lean T2D, which might help to develop better disease prevention and treatment strategies. This review examines the role of protein insufficiency in the maternal diet as the central driver of the developmental programming of lean T2D.

Keywords: Developmental origin of health and disease; Fetal programming; Lean diabetes; Maternal low protein diet; Type 2 diabetes.

©The Author(s) 2022. Published by Baishideng Publishing Group Inc. All rights reserved.

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Conflict of interest statement

Conflict-of-interest statement: Authors have no conflict of interest.

Figures

Figure 1
Figure 1
Key gene expression and epigenetic changes observed in different maternal low protein studies.
Figure 2
Figure 2
Proposed mechanism of maternal low protein associated lean type 2 diabetes.
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References

    1. Report of the Expert Committee on the Diagnosis and Classification of Diabetes Mellitus. Diabetes Care. 1997;20:1183–1197. - PubMed
    1. National diabetes statistics report, 2020. [cited 28 Sep 2021] Available from:https://www.cdc.gov/diabetes/data/statistics-report/index.html .
    1. George AM, Jacob AG, Fogelfeld L. Lean diabetes mellitus: An emerging entity in the era of obesity. World J Diabetes . 2015;6:613–620. - PMC - PubMed
    1. Coleman NJ, Miernik J, Philipson L, Fogelfeld L. Lean versus obese diabetes mellitus patients in the United States minority population. J Diabetes Complications . 2014;28:500–505. - PubMed
    1. Das S, Fonseca V. Low bodyweight type 2 diabetes in India: clinical characteristics and pathophysiology. Diabetes Metab Syndr . 2009;3:60–66.

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