Movatterモバイル変換


[0]ホーム

URL:


 
 
* 611429

APRG1 TUMOR SUPPRESSOR CANDIDATE; APRG1


Alternative titles; symbols

AP20 REGION PROTEIN 1
CHROMOSOME 3 OPEN READING FRAME 35; C3ORF35


HGNC Approved Gene Symbol:APRG1

Cytogenetic location:3p22.2   Genomic coordinates(GRCh38) :3:37,381,077-37,435,497 (from NCBI)


TEXT

Cloning and Expression

By database analysis to identify putative coding sequences in a region of chromosome 3 associated with carcinomas,Protopopov et al. (2003) identified several splice variants of APRG1, which they designated variants A through G. The longest protein, encoded by isoform B, contains 170 amino acids and has a calculated molecular mass of 18.5 kD. It has a putative cell attachment sequence, arg-gly-asp, a C-terminal transmembrane domain, and possible sites for phosphorylation, N-myristoylation, and amidation. Northern blot analysis revealed low abundance of APRG1 mRNA. PCR analysis detected variants B and D highly expressed in pancreas, variant A highly expressed in kidney, and variants B and C highly expressed in placenta.


Gene Function

Leris et al. (2005) found that the level of APRG1 mRNA was lower in malignant breast tissue than in normal tissue. Furthermore, APRG1 expression fell with increasing stage using the tumor-module-metastasis (TNM) classification, and became statistically significant when TNM stages 3 and 4 were compared to TNM stage 1.


Gene Structure

Protopopov et al. (2003) determined that the APRG1 gene contains at least 7 exons and spans about 36.3 kb. There are 2 putative promoter/enhancer sites, the second of which is upstream of exon 2 and contains binding sites for the transcription factors LYF1 (603023), delta-EF1 (189909), TCF11 (163260), GATA (see305371), and others.


Mapping

Using FISH and a deletion cell hybrid panel,Protopopov et al. (2003) mapped the APRG1 gene to chromosome 3p21.3.

Gross (2021) mapped the APRG1 gene to chromosome 3p22.2 based on an alignment of the APRG1 sequence (GenBankBC100861) with the genomic sequence (GRCh38).


REFERENCES

  1. Gross, M. B.Personal Communication. Baltimore, Md. 8/16/2021.

  2. Leris, A. C. A., Roberts, T. R., Jiang, W. G., Newbold, R. F., Mokbel, K.Evidence for a tumour suppressive function of APRG1 in breast cancer. Breast Cancer Res. Treat. 93: 97-100, 2005. [PubMed:16187228,related citations] [Full Text]

  3. Protopopov, A., Kashuba, V., Zabarovska, V. I., Muravenko, O. V., Lerman, M. I., Klein, G., Zabarovsky, E. R.An integrated physical and gene map of the 3.5-Mb chromosome 3p21.3 (AP20) region implicated in major human epithelial malignancies. Cancer Res. 63: 404-412, 2003. [PubMed:12543795,related citations]


Contributors:
Matthew B. Gross - updated : 08/16/2021
Creation Date:
Patricia A. Hartz : 9/13/2007
mgross : 08/16/2021
carol : 06/04/2019
alopez : 09/13/2007

* 611429

APRG1 TUMOR SUPPRESSOR CANDIDATE; APRG1


Alternative titles; symbols

AP20 REGION PROTEIN 1
CHROMOSOME 3 OPEN READING FRAME 35; C3ORF35


HGNC Approved Gene Symbol: APRG1

Cytogenetic location: 3p22.2   Genomic coordinates(GRCh38) : 3:37,381,077-37,435,497(from NCBI)


TEXT

Cloning and Expression

By database analysis to identify putative coding sequences in a region of chromosome 3 associated with carcinomas, Protopopov et al. (2003) identified several splice variants of APRG1, which they designated variants A through G. The longest protein, encoded by isoform B, contains 170 amino acids and has a calculated molecular mass of 18.5 kD. It has a putative cell attachment sequence, arg-gly-asp, a C-terminal transmembrane domain, and possible sites for phosphorylation, N-myristoylation, and amidation. Northern blot analysis revealed low abundance of APRG1 mRNA. PCR analysis detected variants B and D highly expressed in pancreas, variant A highly expressed in kidney, and variants B and C highly expressed in placenta.


Gene Function

Leris et al. (2005) found that the level of APRG1 mRNA was lower in malignant breast tissue than in normal tissue. Furthermore, APRG1 expression fell with increasing stage using the tumor-module-metastasis (TNM) classification, and became statistically significant when TNM stages 3 and 4 were compared to TNM stage 1.


Gene Structure

Protopopov et al. (2003) determined that the APRG1 gene contains at least 7 exons and spans about 36.3 kb. There are 2 putative promoter/enhancer sites, the second of which is upstream of exon 2 and contains binding sites for the transcription factors LYF1 (603023), delta-EF1 (189909), TCF11 (163260), GATA (see 305371), and others.


Mapping

Using FISH and a deletion cell hybrid panel, Protopopov et al. (2003) mapped the APRG1 gene to chromosome 3p21.3.

Gross (2021) mapped the APRG1 gene to chromosome 3p22.2 based on an alignment of the APRG1 sequence (GenBank BC100861) with the genomic sequence (GRCh38).


REFERENCES

  1. Gross, M. B.Personal Communication. Baltimore, Md. 8/16/2021.

  2. Leris, A. C. A., Roberts, T. R., Jiang, W. G., Newbold, R. F., Mokbel, K.Evidence for a tumour suppressive function of APRG1 in breast cancer. Breast Cancer Res. Treat. 93: 97-100, 2005. [PubMed: 16187228] [Full Text: https://doi.org/10.1007/s10549-005-4169-z]

  3. Protopopov, A., Kashuba, V., Zabarovska, V. I., Muravenko, O. V., Lerman, M. I., Klein, G., Zabarovsky, E. R.An integrated physical and gene map of the 3.5-Mb chromosome 3p21.3 (AP20) region implicated in major human epithelial malignancies. Cancer Res. 63: 404-412, 2003. [PubMed: 12543795]


Contributors:
Matthew B. Gross - updated : 08/16/2021

Creation Date:
Patricia A. Hartz : 9/13/2007

Edit History:
mgross : 08/16/2021
carol : 06/04/2019
alopez : 09/13/2007



NOTE: OMIM is intended for use primarily by physicians and other professionals concerned with genetic disorders, by genetics researchers, and by advanced students in science and medicine. While the OMIM database is open to the public, users seeking information about a personal medical or genetic condition are urged to consult with a qualified physician for diagnosis and for answers to personal questions.
OMIM® and Online Mendelian Inheritance in Man® are registered trademarks of the Johns Hopkins University.
Copyright® 1966-2025 Johns Hopkins University.

NOTE: OMIM is intended for use primarily by physicians and other professionals concerned with genetic disorders, by genetics researchers, and by advanced students in science and medicine. While the OMIM database is open to the public, users seeking information about a personal medical or genetic condition are urged to consult with a qualified physician for diagnosis and for answers to personal questions.
OMIM® and Online Mendelian Inheritance in Man® are registered trademarks of the Johns Hopkins University.
Copyright® 1966-2025 Johns Hopkins University.
Printed: Nov. 28, 2025

OMIM Donation:

Dear OMIM User,

To ensure long-term funding for the OMIM project, we have diversified our revenue stream. We are determined to keep this website freely accessible. Unfortunately, it is not free to produce. Expert curators review the literature and organize it to facilitate your work. Over 90% of the OMIM's operating expenses go to salary support for MD and PhD science writers and biocurators. Please join your colleagues by making a donation now and again in the future. Donations are an important component of our efforts to ensure long-term funding to provide you the information that you need at your fingertips.

Thank you in advance for your generous support,
Ada Hamosh, MD, MPH
Scientific Director, OMIM


[8]ページ先頭

©2009-2025 Movatter.jp