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* 608676

TAXILIN, ALPHA; TXLNA


Alternative titles; symbols

TXLN
INTERLEUKIN 14; IL14


HGNC Approved Gene Symbol:TXLNA

Cytogenetic location:1p35.2   Genomic coordinates(GRCh38) :1:32,179,675-32,198,285 (from NCBI)


TEXT

Cloning and Expression

Using a blot overlay technique to identify proteins from rat lung that interact with the SNARE membrane protein Stx3 (600876),Nogami et al. (2003) identified Txln. By searching databases using peptide sequences of purified rat Txln, followed by RACE of a brain cDNA library, they cloned human TXLN. The predicted TXLN protein contains 546 amino acids and has a calculated molecular mass of 62 kD. It has a long C-terminal coiled-coil domain, 24 potential phosphorylation sites, 1 N-glycosylation site, 10 N-myristoylation sites, and 3 EF-hand motifs. Northern blot analysis detected a major 5.1-kb TXLN transcript in all tissues tested, as well as 2.1- and 1.5-kb TXLN transcripts in heart, liver, and pancreas.


Gene Function

Using coimmunoprecipitation experiments,Nogami et al. (2003) showed that TXLN interacts with STX3 through its coiled-coil domain. They found that TXLN also interacts with STX1A (186590) and STX4 (186591). Immunofluorescence and confocal microscopy demonstrated that coexpression of TXLN with STX4 caused relocalization of TXLN from the cytosol to the plasma membrane. Overexpression of TXLN in PC12 cells inhibited human growth hormone (139250) exocytosis in response to high potassium levels.


REFERENCES

  1. Nogami, S., Satoh, S., Nakano, M., Shimizu, H., Fukushima, H., Maruyama, A., Terano, A., Shirataki, H.Taxilin; a novel syntaxin-binding protein that is involved in Ca(2+)-dependent exocytosis in neuroendocrine cells. Genes Cells 8: 17-28, 2003. [PubMed:12558796,related citations] [Full Text]


Creation Date:
Laura L. Baxter : 5/21/2004
carol : 08/27/2014
alopez : 9/14/2007
mgross : 5/21/2004

* 608676

TAXILIN, ALPHA; TXLNA


Alternative titles; symbols

TXLN
INTERLEUKIN 14; IL14


HGNC Approved Gene Symbol: TXLNA

Cytogenetic location: 1p35.2   Genomic coordinates(GRCh38) : 1:32,179,675-32,198,285(from NCBI)


TEXT

Cloning and Expression

Using a blot overlay technique to identify proteins from rat lung that interact with the SNARE membrane protein Stx3 (600876), Nogami et al. (2003) identified Txln. By searching databases using peptide sequences of purified rat Txln, followed by RACE of a brain cDNA library, they cloned human TXLN. The predicted TXLN protein contains 546 amino acids and has a calculated molecular mass of 62 kD. It has a long C-terminal coiled-coil domain, 24 potential phosphorylation sites, 1 N-glycosylation site, 10 N-myristoylation sites, and 3 EF-hand motifs. Northern blot analysis detected a major 5.1-kb TXLN transcript in all tissues tested, as well as 2.1- and 1.5-kb TXLN transcripts in heart, liver, and pancreas.


Gene Function

Using coimmunoprecipitation experiments, Nogami et al. (2003) showed that TXLN interacts with STX3 through its coiled-coil domain. They found that TXLN also interacts with STX1A (186590) and STX4 (186591). Immunofluorescence and confocal microscopy demonstrated that coexpression of TXLN with STX4 caused relocalization of TXLN from the cytosol to the plasma membrane. Overexpression of TXLN in PC12 cells inhibited human growth hormone (139250) exocytosis in response to high potassium levels.


REFERENCES

  1. Nogami, S., Satoh, S., Nakano, M., Shimizu, H., Fukushima, H., Maruyama, A., Terano, A., Shirataki, H.Taxilin; a novel syntaxin-binding protein that is involved in Ca(2+)-dependent exocytosis in neuroendocrine cells. Genes Cells 8: 17-28, 2003. [PubMed: 12558796] [Full Text: https://doi.org/10.1046/j.1365-2443.2003.00612.x]


Creation Date:
Laura L. Baxter : 5/21/2004

Edit History:
carol : 08/27/2014
alopez : 9/14/2007
mgross : 5/21/2004



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OMIM® and Online Mendelian Inheritance in Man® are registered trademarks of the Johns Hopkins University.
Copyright® 1966-2025 Johns Hopkins University.

NOTE: OMIM is intended for use primarily by physicians and other professionals concerned with genetic disorders, by genetics researchers, and by advanced students in science and medicine. While the OMIM database is open to the public, users seeking information about a personal medical or genetic condition are urged to consult with a qualified physician for diagnosis and for answers to personal questions.
OMIM® and Online Mendelian Inheritance in Man® are registered trademarks of the Johns Hopkins University.
Copyright® 1966-2025 Johns Hopkins University.
Printed: March 31, 2025

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