| Eimeria stiedae | |
|---|---|
Scientific classification | |
| Domain: | Eukaryota |
| Clade: | Sar |
| Clade: | Alveolata |
| Phylum: | Apicomplexa |
| Class: | Conoidasida |
| Order: | Eucoccidiorida |
| Family: | Eimeriidae |
| Genus: | Eimeria |
| Species: | E. stiedae |
| Binomial name | |
| Eimeria stiedae (Lindemann, 1895) | |
Eimeria stiedae is a species ofEimeria that causes hepaticcoccidiosis inrabbits.[1] It was observed for the first time byAntonie van Leeuwenhoek in 1674.
The rabbits ingest sporulatedoocysts.[1] Sporulated oocysts containsporozoites that hatch and travel via thehepatic portal vein to the liver, and eventually penetrate thebile ductepithelium, where they undergo asexual reproduction known asschizogony. Rupture of the schizont consequently causes epithelial cell rupture and death. Merozoites will penetrate other cells and create more merozoites for one to several rounds. Eventually, a merozoite becomes a male microgamecyte and asexually reproduces in epithelial cells. A ruptured microgamecyte infects a cell with the female macrogamecyte and through sexual reproduction create azygote. The zygote develops a protective shell before expulsion in the bile excreation and then the feces as an oocyst.[2]
The schizonts in the bile duct cause bile ducthyperplasia, and blockage of bile ducts leading tohepatomegaly and henceicterus. Due to liver failure, the abdomen will be distended with fluid.[2]
The presence of oocysts on fecal flotation or impression smear of the liver are diagnostic forcoccidiosis. The prepatent period is 18 days; sporulation occurs after three days.[1]
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