Anemic infarcts (also calledwhite infarcts orpale infarcts) are white or paleinfarcts caused byarterial occlusions, and are usually seen in the heart, kidney and spleen.
These are referred to as "white" because of the lack of hemorrhaging and limited red blood cells accumulation, (compare toHemorrhagic infarct). The tissues most likely to be affected are solid organs which limit the amount of hemorrhage that can seep into the area of ischemic necrosis from adjoiningcapillary beds. The organs typically include single blood supply (no dual arterial blood supply oranastomoses). The infarct generally results grossly in a wedge shaped area of necrosis with the apex closest to the occlusion and the base at the periphery of the organ. The margins will become better defined with time with a narrow rim of congestion attributable to inflammation at the edge of the lesion.[1] Relatively few extravasated red cells are lysed so the resultinghemosiderosis is limited and results in a progressively paler area of infarction with time. Ischemiccoagulative necrosis results and fibrosis of the affected area develops from the reparative response beginning at the preserved margins and working its way inwards. One exception to coagulative necrosis is the brain, which undergoesliquefactive necrosis in response to infarction.[citation needed]
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