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Abyssinian cat

From Wikipedia, the free encyclopedia
Breed of cat
"Abys" redirects here. For the Swiss politician, seeRageth Abys.

Breed of cat
Abyssinian
A male ruddy Abyssinian
Common nicknamesAbys
OriginIndian Ocean,Southeast Asia[1]
Breed standards
CFAstandard
FIFestandard
TICAstandard
ACFstandard
CCA-AFCstandard
GCCFstandard
Domestic cat (Felis catus)

TheAbyssinian/æbɪˈsɪniən/ is abreed of cat with a distinctive "ticked" tabby coat, in which individual hairs are banded with different colours.[2] They are also known simply asAbys.[3]

The first members of the breed to be exhibited in England were brought there fromAbyssinia (now known asEthiopia), hence the name. Genetic studies place the breed's origins inSoutheast Asia and the coasts of theIndian Ocean, however. It is possible that the breed was introduced to Abyssinia by travelers who had stopped inCalcutta.[4]

Once a comparatively obscure breed, the Abyssinian had become one of the top five most popular cat breeds by 2016.[3]

The breed's distinctive appearance, seeming long, lean and finely coloured compared to other cats, has been analogized to that of humanfashion models. Personality-wise, the cats traditionally display active, curious attitudes in which they frequently follow owners around and encourage play.[3] Theirdog-like characteristics also involve a particular sense of affection and desire for interaction. Abys have a distinctive wildcat look with their ticked coat and large erect ears. They are a highly social breed and can be demanding of attention. They do well in multi-cat households due to their social nature. Not a lap cat, Abyssinians are in constant motion, either exploring or playing.[5]

History

[edit]
Illustration of the so-called first abyssinian cat, Zula, from the book by Dr. W. Gordon Stables: Cats, Their Points and Characteristics and Curiosities of Cat Life, 1874
Zula, the so-called "first Abyssinian"

What is thought to be the earliest known designated Abyssinian cat is in an exhibit still residing in the Leiden Zoological Museum inThe Netherlands. It was purchased around 1834-1836 from a supplier of small wild cat exhibits as a taxidermy and was labeled by the museum founder as "Patrie, domestica India."[6] The first example of a domesticated Abyssinian, however, involves the story of a cat being brought to England by the BritishLt. General Sir Robert Napier in 1868 who had returned from theAbyssinia War. The cat was given the name "Zula" and won first prize in the December 1871Crystal Palace cat show.[6][7]

Many modern Abyssinian breeders dispute Zula as having been the first domestic Abyssinian, arguing that the existing illustrations of Zula portray the cat as having ears too small for an Abyssinian and a coat too waved and long.

The breed was nearly wiped out in theUnited Kingdom following theSecond World War and an outbreak offeline leukaemia virus, resulting in cats being imported from places such as Holland, America,Scandinavia,Australia, andNew Zealand.[8]

The Abyssinian is one of the oldest established cat breeds, being recognised in 1929 by theGoverning Council of the Cat Fancy. The breed was developed in the United Kingdom with references dating back to the 1890s.[8][9]

Description

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Appearance

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The Abyssinian is a lithe, fine-boned, muscular, medium to large sized cat. The average weight is 10 lb (4.5 kg) ranging between 8–12 lb (3.6–5.4 kg) with height ranging between 8–10 in (20–25 cm).[9][10] The head is moderately wedge-shaped, with a slight break at the muzzle, and nose and chin ideally forming a straight vertical line when viewed in profile. They have alert, relatively large pointed ears. The eyes are almond-shaped and are gold, green, hazel or copper depending on coat colour. The legs tend to be long in proportion to a graceful body, with small oval paws; the tail is likewise long and tapering.[11]

Close-up view of the fur of an Abyssinian cat, showing the "ticked" effect
The Abyssinian's fur exhibits a "ticked" effect.

Abyssinian kittens are born with dark coats that gradually lighten as they mature, usually over several months.[citation needed] The coat is short, and is ideally fine, not soft, dense, close-lying and silky to the touch. The ticked oragouti effect that is the trademark of the breed—genetically a variant of thetabby pattern—should be uniform over the body, although the ridge of the spine and tail, back of the hind legs and the pads of the paws are always noticeably darker. Each hair has a light base with three or four bands of additional colour growing darker towards the tip. The base colour should be as clear as possible; any extensive intermingling with grey is considered a serious fault. A tendency to white on the chin is common but likewise must be minimal. The typical tabby M-shaped marking is often found on the forehead.[9][11]

Photograph of a champion adult male Abyssinian cat, showing the classic ruddy coat pattern
A champion adult male showing the classic ruddy, or "usual", coat pattern

The breed's original colour standard is a warm deep reddish-brown base with black ticking, known as "usual" in the United Kingdom, "tawny" in Australia, and "ruddy" elsewhere. Sorrel (also called cinnamon or red), a lighter coppery base with chocolate brown ticking, is a unique mutation of this original pattern. Other variants have been introduced by outcrossing to theBurmese and other shorthaired breeds, notably blue (on a warm beige base) and fawn (on a softer creamy peach base). The less common chocolate and lilac are not recognized in the Cat Fancier's Association (CFA) breed standard[12] but have been granted full champion status in The International Cat Association (TICA)[13] and in the UK. The UK also recognizes the Silver Abyssinian, in which the base coat is a pure silvery white with black (called "usual silver"), blue, cream or sorrel ticking. Various other colour combinations are in development, including the "torbie", in which a patchedtortoiseshell pattern in any of these colours is visible under the tabby banding.

The breed owes their distinctive coat to a dominant mutant gene known as Ta. In 2007, the first cat to have its entire genome published was an Abyssinian named Cinnamon.[14]

Behaviour

[edit]
Photograph of three Abyssinian kittens, showing three different coat colours: ruddy, fawn, and blue
Abyssinian kittens. Clockwise from bottom left: ruddy, fawn and blue.

Veterinarian Joan O. Joshua has written that the "dog-like attachment to the owners" of Abyssinian andBurmese cats causes "greater dependence on human contacts". This stands in contrast to the mere "tolerant acceptance of human company" based around "comforts" that multiple other breeds display.[5]

With their interest in playing with their owners combined with their curious intelligence, Abyssinians are sometimes called the "Clowns of the Cat Kingdom".[3] They have soft chirrup-like vocalizations which do not sound like the expected "meow".[citation needed]

A study comparingOriental,Siamese and Abyssinian kittens toNorwegian Forest cat kittens found that the former group was more likely to recede and hide as well as display other 'shy' behaviour.[15]

Health

[edit]

Familial renal amyloidosis orAA amyloidosis, a kidney disorder due to a mutation in the AA amyloid protein gene, has been seen in Abyssinians.[16] The Abyssinian has had severe problems with blindness caused by a hereditary retinal degeneration due to mutations in the rdAc gene. However, the prevalence has been reduced from 45% to less than 4% in 2008 in the country of Sweden.[17] An Australian analysis found the Abyssinian to be over-represented in cases offeline infectious peritonitis when compared to the expected frequency based on census data (4.4% versus 1.5%).[18] An American study had similar results with an odds ratio of 8.98.[19]

In a review of over 5,000 cases ofurate urolithiasis the Abyssinian was significantly under-represented, with only one of the recorded cases belonging to an Abyssinian.[20]

The 2008 study "The Ascent of Cat Breeds: Genetic Evaluations of Breeds and Worldwide Random-bred Populations" by Lipinski et al. conducted at UC Davis by the team led by leading feline geneticist Dr Leslie Lyons found that the Abyssinian has a low level of genetic diversity, a heterozygosity value of 0.45 within a range of 0.34–0.69 for all breeds studied, and has genetic markers common to both Southeast Asian and Western breeds indicating that cats from both Asia and Europe were used to create the breed.[21]

The Abyssinian was found to be predisposed tofeline atopic dermatitis in a retrospective study of cases of the disease.[22]

The Abyssinian is predisposed topsychogenic alopecia.[23]

An American study found the Abyssinian to be at increased risk ofaorticthromboembolism with an odds ratio of 6.03.[19]

A retrospective study in the US found the Abyssinian to be predisposed to acquiredmyasthaenia gravis with an odds ratio of 4.97.[19]

Mycobacterium avium complex infection is a very rare disease: 10/12 cases were Abyssinians.[19]

A study of cases ofpatellar luxation in the USA and in Europe found 38% (26/69) Abyssinians had the condition compared to 1/84 for other breeds.[19]

The Abyssinian is the cat breed most commonly affected byprogressive retinal atrophy. The condition is caused by two separate mutations in the breed. Early onset PRA is caused by anautosomal dominant mutation in theCRX gene. Late onset PRA is caused by anautosomal recessive mutation in theCEP290 gene.[24]

The Abyssinian is one of the more commonly affected breeds forpyruvate kinase deficiency. Anautosomal recessive mutation of thePKLR gene is responsible for the condition in the breed.[24]

See also

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References

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  1. ^"About the Abyssinian".www.cfa.org.Archived from the original on 22 April 2023. Retrieved11 October 2024.
  2. ^"Abyssinian Breed Description".catzinc.org. Retrieved31 January 2018.
  3. ^abcd"Abyssinian".VCA Animal Hospitals. Retrieved9 September 2016.
  4. ^"About the Abyssinian". Archived fromthe original on 5 January 2013. Retrieved31 January 2018.
  5. ^abJoshua, Joan O. (2013).The Clinical Aspects of Some Diseases of Cats. Elsevier. p. 1.ISBN 9781483226002.
  6. ^ab"Abyssinian". 23 July 2013.
  7. ^"All About Abyssinian Cats - History of the Abyssinian Cat".
  8. ^abBarnes-Hookey, Jacquie."History of the Abyssinian Cat".Abyssinian Cat Association. Retrieved1 January 2024.
  9. ^abc"Abyssinian".Governing Council of the Cat Fancy. Retrieved1 January 2024.
  10. ^Leeson, Janelle."Abyssinian".PetMD. Retrieved3 January 2024.
  11. ^ab"Abysinnian standard".Abyssinian Cat Club. Retrieved3 January 2024.
  12. ^"Abyssinian : POINT SCORE"(PDF).Cfainc.org. Archived fromthe original(PDF) on 1 July 2014. Retrieved15 December 2017.
  13. ^"Abyssinian Breed Group Standard"(PDF).tica.org. 1 May 2008. Retrieved11 October 2024.
  14. ^Highfield, Roger (31 October 2007)."Cinnamon the cat could offer hope to the blind". The Daily Telegraph.Archived from the original on 3 November 2007. Retrieved26 June 2023.
  15. ^Marchei, P.; Diverio, S.; Falloci, N.; Fatjó, J.; Ruiz-de-la-Torre, J. L.; Manteca, X. (23 March 2009)."Breed differences in behavioural development in kittens".Physiology & Behavior.96 (4–5):522–531.doi:10.1016/j.physbeh.2008.11.015.S2CID 12358514. Retrieved6 January 2024.
  16. ^Niewold TA, van der Linde-Sipman JS, Murphy C, Tooten PC, Gruys E (September 1999). "Familial amyloidosis in cats: Siamese and Abyssinian AA proteins differ in primary sequence and pattern of deposition".Amyloid.6 (3):205–9.doi:10.3109/13506129909007328.PMID 10524286.
  17. ^"Theodosius Dobzhansky Center for Genome Bioinformatics"(PDF).Dobzhanskycenter.bio.spbu.ru. Archived fromthe original(PDF) on 17 December 2013. Retrieved15 December 2017.
  18. ^Worthing, Kate A; Wigney, Denise I; Dhand, Navneet K; Fawcett, Anne; McDonagh, Phillip; Malik, Richard; Norris, Jacqueline M (7 March 2012)."Risk factors for feline infectious peritonitis in Australian cats".Journal of Feline Medicine and Surgery.14 (6):405–412.doi:10.1177/1098612X12441875.hdl:2123/14691.PMID 22398460. Retrieved7 January 2024.
  19. ^abcdeGough, Alex; Thomas, Alison; O'Neill, Dan G. (2018).Breed Predispositions to Disease in Dogs and Cats. Hoboken: Wiley Blackwell. pp. 227–228.ISBN 978-1-119-22558-4.
  20. ^Albasan, H.; Osborne, C. A.; Lulich, J. P.; Lekcharoensuk, C. (2012)."Risk factors for urate uroliths in cats".Journal of the American Veterinary Medical Association.240 (7):842–847.doi:10.2460/javma.240.7.842.PMID 22443437.
  21. ^Lipinski MJ, Froenicke L, Baysac KC, Billings NC, Leutenegger CM, Levy AM, Longeri M, Niini T, Ozpinar H, Slater MR, Pedersen NC, Lyons LA (January 2008)."The ascent of cat breeds: genetic evaluations of breeds and worldwide random-bred populations".Genomics.91 (1):12–21.doi:10.1016/j.ygeno.2007.10.009.PMC 2267438.PMID 18060738.
  22. ^Ravens, Philippa A.; Xu, Bei J.; Vogelnest, Linda J. (2014). "Feline atopic dermatitis: a retrospective study of 45 cases (2001–2012)".Veterinary Dermatology.25 (2): 95.doi:10.1111/vde.12109.ISSN 0959-4493.
  23. ^Hnilica, Keith A.; Patterson, Adam P. (19 September 2016).Small Animal Dermatology. St. Louis (Miss.): Saunders.ISBN 978-0-323-37651-8.
  24. ^abOliver, James A.C.; Mellersh, Cathryn S. (2020). "Genetics". In Cooper, Barbara; Mullineaux, Elizabeth; Turner, Lynn (eds.).BSAVA Textbook of Veterinary Nursing (Sixth ed.). British Small Animal Veterinary Association. p. 131.ISBN 978-1-910-44339-2.

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